Anabolic Steroids - Steroidal.com Forums - Powered by vBulletin
Connect with us on Facebook Follow us on Twitter Follow us on Google Plus
Steroids For Sale
Results 1 to 26 of 26
Like Tree1Likes
  • 1 Post By Bull

Thread: Post-Workout Protocol

  1. #1
    Member Bull's Avatar
    Join Date
    Apr 2013
    Location
    Canada
    Posts
    371

    Post-Workout Protocol

    What's everyone's post workout protocol? Drugs and nutrition. I'll start.

    When I get to my car: 100mcg MOD GRF 1-29,100mcg GHRP-2 and 7iu humulin-r(or humalog, wait 10 min after peptides for humalog)
    20 minutes later: 1 scoop whey, 5g glutamine, 5g creatine, 2g beta-alanine, 5gbcaas, and 35g simple carbs

    After that I'll have a low fat, carb rich meal. Usually oatmeal and whey with a pack of those flavored oats thrown in for the simple carbs.

    Days with no insulin is the same except peptides are pre workout.

  2. #2
    Administrator Admin's Avatar
    Join Date
    Mar 2013
    Posts
    1,739
    Quote Originally Posted by Bull View Post
    What's everyone's post workout protocol? Drugs and nutrition. I'll start.

    When I get to my car: 100mcg MOD GRF 1-29,100mcg GHRP-2 and 7iu humulin-r(or humalog, wait 10 min after peptides for humalog)
    20 minutes later: 1 scoop whey, 5g glutamine, 5g creatine, 2g beta-alanine, 5gbcaas, and 35g simple carbs

    After that I'll have a low fat, carb rich meal. Usually oatmeal and whey with a pack of those flavored oats thrown in for the simple carbs.

    Days with no insulin is the same except peptides are pre workout.
    PWO was usually Whey Iso with added Leucine, carbs.

    Intra was PeptoPro and/or BCAAs.

    Solid meal 1 hour after training, usually fish, rice, vegetables.

    If I was using peptides, CJC 1295 100mcg with GHRP-6 100mcg PWO.

  3. #3
    Junior Member
    Join Date
    Oct 2013
    Posts
    10
    One Chipotle burrito with white rice, black beans, chicken, hot sauce, cheese and lettuce about 30 mins post workout.

  4. #4
    Member Bull's Avatar
    Join Date
    Apr 2013
    Location
    Canada
    Posts
    371
    Quote Originally Posted by 3Osos View Post
    One Chipotle burrito with white rice, black beans, chicken, hot sauce, cheese and lettuce about 30 mins post workout.
    Damn, that sounds a lot better than my oatmeal!

  5. #5
    Member OnTheSauce's Avatar
    Join Date
    Nov 2013
    Posts
    460
    You should probably wait before hitting the slin shot while using peptides. Insulin would blunt the gh release from the peptides

  6. #6
    Member Bull's Avatar
    Join Date
    Apr 2013
    Location
    Canada
    Posts
    371
    Quote Originally Posted by OnTheSauce View Post
    You should probably wait before hitting the slin shot while using peptides. Insulin would blunt the gh release from the peptides
    It's not the insulin per-say, it's the ingestion of carbs/fats that blunt it. Even so, the blunt would only be 80% at most. I can't be arsed to look through hundreds of bookmarks for the studies right now, but it's fine if they're in there together. I do wait 20+ minutes before taking in any sugars though after my GHRP-MOD shots, as that's when the pulse starts to drop.

  7. #7
    Member OnTheSauce's Avatar
    Join Date
    Nov 2013
    Posts
    460
    When you ingest carbs, what happens? Insulin spikes. That is why. Insulin blunts gh and vice versa

  8. #8
    Member Bull's Avatar
    Join Date
    Apr 2013
    Location
    Canada
    Posts
    371
    Quote Originally Posted by OnTheSauce View Post
    When you ingest carbs, what happens? Insulin spikes. That is why. Insulin blunts gh and vice versa
    That's bro science. I have more studies similar kicking around here somewhere.


    Effect of Growth hormone (GH)-releasing hormone (GHRH), atropine, pyridostigmine, or hypoglycemia on GHRP-6-induced GH secretion in man


    A Penalva, A Carballo, M Pombo, FF Casanueva and C Dieguez
    Department of Medicine, Faculty of Medicine, University of Santiago, Santiago de Compostela, Spain. His-DTrp-Ala-Trp-DPhe-Lys-NH2 (GHRP-6) is a synthetic compound that releases GH in a dose-related and specific manner in several species, including man. To further characterize the effects and mechanism of action of GHRP-6 on GH secretion, we assessed in normal man plasma GH responses to that hexapeptide 1) alone and in combination with exogenous GH-releasing hormone (GHRH) administration, 2) in a state of high endogenous somatostatinergic tone after atropine administration, and 3) in a state of low endogenous somatostatinergic tone induced by the cholinergic receptor agonist drug pyridostigmine or after insulin- induced hypoglycemia. We found a similar increase in plasma GH levels after the administration of either GHRP-6 (1 microgram/kg) or GHRH (1 microgram/kg); the areas under the curve (AUC) were (mean +/- SEM) 973 +/- 181 and 821 +/- 139, respectively. After combined GHRP-6 and GHRH administration, GH responses were considerably greater than those after either compound alone (4412 +/- 842; P < 0.01). Administration of the cholinergic receptor antagonist atropine (1 mg, im) completely prevented the GH responses to GHRP-6 (area under the curve, 103 +/- 14 vs. 815 +/- 156, respectively). On the other hand, pyridostigmine, a cholinergic agonist, slightly increased GH responses to GHRP-6 (P < 0.01 when comparing the AUC after pyridostigmine administration of 1571 +/- 151 and the AUC after administration of GHRP-6 alone of 815 +/- 156). Finally, combined GHRP-6 and insulin administration induced a much greater increase in plasma GH levels (AUC, 4047 +/- 327) than insulin alone (1747 +/- 229; P < 0.05) or GHRP-6 alone (1248 +/- 376; P < 0.05). Our results lend support to the view that GHRP-6-induced GH secretion is exerted through a non-GHRH-dependent mechanism. Furthermore, the fact that enhancement of somatostatinergic tone with atropine completely prevented the GH responses to GHRP-6, while pyridostigmine and insulin-induced hypoglycemia, which increased plasma GH levels by inhibiting hypothalamic somatostatin release, increased the same response suggest that although GHRP-6-induced GH secretion is dependent on the endogenous somatostatinergic tone, the stimulatory effect of GHRP-6 on plasma GH levels is not mediated by a change in hypothalamic somatostatinergic tone.
    Admin likes this.

  9. #9
    Junior Member DrBPackenwood's Avatar
    Join Date
    Apr 2013
    Location
    Australia
    Posts
    84
    i go home, eat, shower, then take a nap

    most of the time...

  10. #10
    Administrator Admin's Avatar
    Join Date
    Mar 2013
    Posts
    1,739
    Quote Originally Posted by Bull View Post
    That's bro science. I have more studies similar kicking around here somewhere.


    Effect of Growth hormone (GH)-releasing hormone (GHRH), atropine, pyridostigmine, or hypoglycemia on GHRP-6-induced GH secretion in man


    A Penalva, A Carballo, M Pombo, FF Casanueva and C Dieguez
    Department of Medicine, Faculty of Medicine, University of Santiago, Santiago de Compostela, Spain. His-DTrp-Ala-Trp-DPhe-Lys-NH2 (GHRP-6) is a synthetic compound that releases GH in a dose-related and specific manner in several species, including man. To further characterize the effects and mechanism of action of GHRP-6 on GH secretion, we assessed in normal man plasma GH responses to that hexapeptide 1) alone and in combination with exogenous GH-releasing hormone (GHRH) administration, 2) in a state of high endogenous somatostatinergic tone after atropine administration, and 3) in a state of low endogenous somatostatinergic tone induced by the cholinergic receptor agonist drug pyridostigmine or after insulin- induced hypoglycemia. We found a similar increase in plasma GH levels after the administration of either GHRP-6 (1 microgram/kg) or GHRH (1 microgram/kg); the areas under the curve (AUC) were (mean +/- SEM) 973 +/- 181 and 821 +/- 139, respectively. After combined GHRP-6 and GHRH administration, GH responses were considerably greater than those after either compound alone (4412 +/- 842; P < 0.01). Administration of the cholinergic receptor antagonist atropine (1 mg, im) completely prevented the GH responses to GHRP-6 (area under the curve, 103 +/- 14 vs. 815 +/- 156, respectively). On the other hand, pyridostigmine, a cholinergic agonist, slightly increased GH responses to GHRP-6 (P < 0.01 when comparing the AUC after pyridostigmine administration of 1571 +/- 151 and the AUC after administration of GHRP-6 alone of 815 +/- 156). Finally, combined GHRP-6 and insulin administration induced a much greater increase in plasma GH levels (AUC, 4047 +/- 327) than insulin alone (1747 +/- 229; P < 0.05) or GHRP-6 alone (1248 +/- 376; P < 0.05). Our results lend support to the view that GHRP-6-induced GH secretion is exerted through a non-GHRH-dependent mechanism. Furthermore, the fact that enhancement of somatostatinergic tone with atropine completely prevented the GH responses to GHRP-6, while pyridostigmine and insulin-induced hypoglycemia, which increased plasma GH levels by inhibiting hypothalamic somatostatin release, increased the same response suggest that although GHRP-6-induced GH secretion is dependent on the endogenous somatostatinergic tone, the stimulatory effect of GHRP-6 on plasma GH levels is not mediated by a change in hypothalamic somatostatinergic tone.
    Good post.

  11. #11
    Member OnTheSauce's Avatar
    Join Date
    Nov 2013
    Posts
    460
    I take hgh and my insulin sensitivity is SMASHED. My blood sugar sky rockets. That's not bro science. That's very first hand experience.

    How it's affected with ghrp I can't comment

  12. #12
    Member Bull's Avatar
    Join Date
    Apr 2013
    Location
    Canada
    Posts
    371
    Quote Originally Posted by OnTheSauce View Post
    I take hgh and my insulin sensitivity is SMASHED. My blood sugar sky rockets. That's not bro science. That's very first hand experience.

    How it's affected with ghrp I can't comment
    I have a study somewhere on that. Exogenous HGH over time causes insulin resistance, eventually completely inhibiting insulin's effect, which is why so many bodybuilders need to use high doses of insulin to continue getting anything from it, and why some develop diabetes with it's use. Can't find it for the life of me though.

    Edit:
    Here we are:

    http://press.endocrine.org/doi/abs/1...jcem-54-5-1033

    Both Human Pituitary Growth hormone and Recombinant DNA-Derived Human Growth Hormone Cause Insulin Resistance at a Postreceptor Site

    RON G. ROSENFELD†, DARRELL M. WILSON, LAURA A. DOLLAR, ANN BENNETT, and RAYMOND L. HINTZ

    We have investigated the effects on carbohydrate metabolism of human GH produced by recombinant DNA technology (methionyl-hGH) compared with pituitary hGH, Twelve normal adult male subjects received four daily im injections of either methionyl-hGH or pituitary hGH in a double blind, crossover study. Oral glucose tolerance tests and assays of insulin binding to peripheral monocytes were performed before the initial administration and 12 h after the fourth injection of both hGH preparations.
    Both methionyl-hGH and pituitary hGH resulted in significant carbohydrate intolerance, with a rise in fasting plasma glucose from 96.6 2.9 to 105.9 3.0 mg/ml (mean SEM) after pituitary hGH and from 96.2 1.5 to 107.5 3.3 mg/dl after methionyl-hGH (P < 0.01). The area under the glucose tolerance curve increased by 34% after pituitary hGH and by 37% after methionyl-hGH. With both hGH preparations, carbohydrate intolerance was associated with marked hyperinsulinemia, with a rise in fasting plasma insulin levels from 9.4 1.2 to 33.2 7.8 U/ml after pituitary hGH and from 7.4 1.1 to 45.8 11.1 U/ml after methionyl-hGH (P < 0.01). The integrated plasma insulin levels during the oral glucose tolerance test tripled after both hGH preparations.
    The pronounced insulin resistance could not be attributed to an alteration in insulin receptor concentrations. Both hGH preparations were associated with small reductions in insulin binding o t monocytes at tracer concentrations, but the decline in binding was not statistically significant. The calculated binding sites per cell and K were not significantly altered by hGH administration.
    We conclude that methionyl-hGH and pituitary hGH are indistinguishable in their ability to induce insulin-resistant carbohydrate intolerance. This decrease in insulin sensitivity cannot be attributed to an alteration in insulin binding, and presumably represents a postreceptor defect in insulin action. (J Clin En-docrinol Metab 54: 1033, 1982)
    Last edited by Bull; 02-27-2014 at 11:27 PM.

  13. #13
    Member OnTheSauce's Avatar
    Join Date
    Nov 2013
    Posts
    460
    I'm already type 2 so yeah I see that. I've taken 40iu and had it do nothing before. Kinda scary. I take a month or two off gh every year. I'm considering only using it in low doses. The stress and everything else makes it worse

  14. #14
    Administrator Admin's Avatar
    Join Date
    Mar 2013
    Posts
    1,739
    Quote Originally Posted by OnTheSauce View Post
    I'm already type 2 so yeah I see that. I've taken 40iu and had it do nothing before. Kinda scary. I take a month or two off gh every year. I'm considering only using it in low doses. The stress and everything else makes it worse
    What brand of HGH do you use?

    What Chinese HGH have you used?

  15. #15
    Member Bull's Avatar
    Join Date
    Apr 2013
    Location
    Canada
    Posts
    371
    Quote Originally Posted by OnTheSauce View Post
    I'm already type 2 so yeah I see that. I've taken 40iu and had it do nothing before. Kinda scary. I take a month or two off gh every year. I'm considering only using it in low doses. The stress and everything else makes it worse
    That's very scary stuff man.

  16. #16
    VET warmouth's Avatar
    Join Date
    Apr 2013
    Location
    Georgia
    Posts
    1,746
    50 grams whey isolate about 30minutes
    after. Then dinner within an hour consisting of approximately 80grams protien/trace-30carbs (depending on the days consumption)/20 grams of fat. Thats it, then 2 snacks high protien/high fat before bed (no carbs).

  17. #17
    Junior Member Phil C's Avatar
    Join Date
    Feb 2014
    Location
    Canada
    Posts
    13
    Typical Post Workout for me is:

    150mcg GHRP-6
    150mcg cjc 1295 no dac
    10iu Humalog

    30 min after pinning the above, a shake consisting of 50g Vitargo, 50g Dextrose, 60g whey protein

    Dinner, 1 hr post shake: High protein and carbs...approx. 60g protein and 80g carbs, minimal fats.

    My last three weeks of dinners have consisted of 200g Tilapia cooked with only a little Pam on the cooking sheet to keep from sticking in the oven, 2/3 cup of natural granola mixed with 2 non fat greek yogurts and half a kilo of blueberries.

    This is my favourite meal and I'm one of those guys that doesn't mind eating the same thing everyday, lol. Every so often I'll swap Tilapia for chicken breast but by far Tilapia is my fave.

  18. #18
    Member OnTheSauce's Avatar
    Join Date
    Nov 2013
    Posts
    460
    Quote Originally Posted by Admin View Post
    What brand of HGH do you use?

    What Chinese HGH have you used?
    Serostim

  19. #19
    VET warmouth's Avatar
    Join Date
    Apr 2013
    Location
    Georgia
    Posts
    1,746
    To the guys that eat tilapia, GROSS! Fish is my favorite food on the planet, but after watching how tilapia is farmed, hell to the no.

  20. #20
    Administrator Admin's Avatar
    Join Date
    Mar 2013
    Posts
    1,739
    We wrote about the importance of Pre-WO nutrition a couple of days ago.


    Whey Protein Pre-Workout Reduces Cortisol & Increases Testosterone | Steroidal.com

  21. #21
    Junior Member luckyfd's Avatar
    Join Date
    Apr 2014
    Posts
    46
    this has always been a huge question for me... I take powdered gatoraid approc 21g dextrose, heaping scoop creatine monohydrate approx.
    5-7g, and 5g bcaas then about 15 min later approx. 50g whey protein isolate. is this completely off? if so id like to fix!!! id hate to be wasting that "window".

  22. #22
    Member Bull's Avatar
    Join Date
    Apr 2013
    Location
    Canada
    Posts
    371
    Quote Originally Posted by luckyfd View Post
    this has always been a huge question for me... I take powdered gatoraid approc 21g dextrose, heaping scoop creatine monohydrate approx.
    5-7g, and 5g bcaas then about 15 min later approx. 50g whey protein isolate. is this completely off? if so id like to fix!!! id hate to be wasting that "window".
    Looks good to me man.

  23. #23
    Junior Member luckyfd's Avatar
    Join Date
    Apr 2014
    Posts
    46
    ok, thanks. maybe I was misunderstanding the suppressing of GH post workout...

  24. #24
    Administrator Admin's Avatar
    Join Date
    Mar 2013
    Posts
    1,739
    Quote Originally Posted by Bull View Post
    Looks good to me man.
    Yes, nothing wrong with that.

    Looks good.

    There is probably a thousand ways to skin the cat when it comes to pre-WO, intra-WO and PWO nutrition.

  25. #25
    Member Bull's Avatar
    Join Date
    Apr 2013
    Location
    Canada
    Posts
    371
    Quote Originally Posted by luckyfd View Post
    ok, thanks. maybe I was misunderstanding the suppressing of GH post workout...
    Oh, in that case... Wait 30 minutes before you have any carbs, protein, or fat. The post workout endogenous GH spike beings to fall around the 25-30 minute mark.

  26. #26
    Junior Member luckyfd's Avatar
    Join Date
    Apr 2014
    Posts
    46
    yes, theres always a diff point of view...thanks again

Posting Permissions

  • You may not post new threads
  • You may not post replies
  • You may not post attachments
  • You may not edit your posts
  •